Search results for "Chinese hamster"

showing 10 items of 78 documents

Transient Multivalent Nanobody Targeting to CD206-Expressing Cells via PH-Degradable Nanogels

2020

To target nanomedicines to specific cells, especially of the immune system, nanobodies can be considered as an attractive tool, as they lack the Fc part as compared to traditional antibodies and, thus, prevent unfavorable Fc-receptor mediated mistargeting. For that purpose, we have site-specifically conjugated CD206/MMR-targeting nanobodies to three types of dye-labeled nanogel derivatives: non-degradable nanogels, acid-degradable nanogels (with ketal crosslinks), and single polymer chains (also obtained after nanogel degradation). All of them can be obtained from the same reactive ester precursor block copolymer. After incubation with na&iuml

0301 basic medicineEndosomeNanogels02 engineering and technologyConjugated systemArticleM2 macrophage03 medical and health sciencesHumansReversible addition−fragmentation chain-transfer polymerizationlcsh:QH301-705.5targetingchemistry.chemical_classificationRAFT polymerizationChinese hamster ovary cellGeneral MedicinePolymerHydrogen-Ion Concentrationmultivalency021001 nanoscience & nanotechnologynanobody030104 developmental biologyTAMchemistryCD206lcsh:Biology (General)nanogelclick chemistryClick chemistryBiophysicsNanocarriers0210 nano-technologyNanogelCells
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The IgGFc-binding protein FCGBP is secreted with all GDPH sequences cleaved but maintained by interfragment disulfide bonds

2021

Mucus forms an important protective barrier that minimizes bacterial contact with the colonic epithelium. Intestinal mucus is organized in a complex network with several specific proteins, including the mucin-2 (MUC2) and the abundant IgGFc-binding protein, FCGBP. FCGBP is expressed in all intestinal goblet cells and is secreted into the mucus. It is comprised of repeated von Willebrand D (vWD) domain assemblies, most of which have a GDPH amino acid sequence that can be autocatalytically cleaved, as previously observed in the mucins MUC2 and mucin-5AC. However, the functions of FCGBP in the mucus are not understood. We show that all vWD domains of FCGBP with a GDPH sequence are cleaved and …

0301 basic medicineMUC5AC mucin-5ACMUC2 mucin-2 (Muc2 mouse)vWF von Willebrand factorBiochemistryvon Willebrand domainchemistry.chemical_compoundPVDF polyvinylidene difluorideMiceCricetinaeDisulfidesIntestinal MucosaPeptide sequenceEndoH endoglycosidase HbiologyChemistryrespiratory systemGDPH Gly-Asp-Pro-HisChaotropic agentBiochemistryWB Western blotIodoacetamideGuHCl guanidinium chlorideResearch ArticleIgG immunoglobulin GvWD von Willebrand D domainCHO CellsCHO Chinese hamster ovary03 medical and health sciencesEndoglycosidase HCricetulusProtein Domainsmucusvon Willebrand FactorAnimalsHumansintestinal epitheliumMolecular BiologyintestineFCGBP IgGFc-binding protein (Fcgbp mouse)GAPH Gly-Ala-Pro-HisMucin-2030102 biochemistry & molecular biologycolonBinding proteinEndoplasmic reticulumMucinITH3 inter-alpha-trypsin inhibitor heavy chain 3Cell BiologyMucusMice Inbred C57BL030104 developmental biologyMUC2Proteolysisbiology.proteinImmunoglobulin G (IgG)IAA iodoacetamideCell Adhesion MoleculesdisulfideThe Journal of Biological Chemistry
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Agonist‐induced desensitisation of β 3 ‐adrenoceptors: Where, when, and how?

2019

β3 -Adrenoceptor agonists have proven useful in the treatment of overactive bladder syndrome, but it is not known whether their efficacy during chronic administration may be limited by receptor-induced desensitisation. Whereas the β2 -adrenoceptor has phosphorylation sites that are important for desensitisation, the β3 -adrenoceptor lacks these; therefore, it had been assumed that β3 -adrenoceptors are largely resistant to agonist-induced desensitisation. While all direct comparative studies demonstrate that β3 -adrenoceptors are less susceptible to desensitisation than β2 -adrenoceptors, desensitisation of β3 -adrenoceptors has been observed in many models and treatment settings. Chimeric …

0301 basic medicinePharmacologyAgonistMessenger RNAmedicine.medical_specialtyCell typePhosphorylation sitesAdrenergic receptormedicine.drug_classbusiness.industryChinese hamster ovary cellTransfection03 medical and health sciences030104 developmental biology0302 clinical medicineEndocrinologyInternal medicinemedicinebusinessReceptor030217 neurology & neurosurgeryBritish Journal of Pharmacology
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Mechanisms of beauvericin toxicity and antioxidant cellular defense

2015

Beauvericin (BEA) is a secondary metabolite produced by many species of fungus Fusarium. This study determines the injury (cell viability, cell proliferation, mitochondrial membrane potential, cell death and DNA damage) and the intracellular defense mechanisms (catalase and superoxide dismutase) in Chinese Hamster ovary (CHO-K1) cells after BEA exposure. The results obtained in this study demonstrated that BEA induces cytotoxicity in a dose- and time-dependent manner in CHO-K1 cells. Moreover, disruption in mitochondrial enzymatic activity and cell proliferation has been observed after BEA exposure, which can lead or be consequence of cell death. BEA inhibits cell proliferation by arresting…

0301 basic medicineProgrammed cell deathCell SurvivalDNA damageApoptosisCHO CellsToxicologyAntioxidantsSuperoxide dismutase03 medical and health sciencesCricetulus0404 agricultural biotechnologyDepsipeptidesAnimalsViability assayCell ProliferationMembrane Potential MitochondrialbiologySuperoxide DismutaseCell growthChinese hamster ovary cell04 agricultural and veterinary sciencesGeneral MedicineCatalase040401 food scienceCell biology030104 developmental biologyBiochemistryApoptosisbiology.proteinIntracellularDNA DamageToxicology Letters
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Intestinal CD36. A lipid-sensor involved in the processing of chylomicrons in rodents

2011

International audience; CD36 is a multifunctional glycoprotein which binds nanomolar concentrations of long-chain fatty acids (LCFA) and is highly expressed on the luminal surface of enterocytes. Despite of its implication in oleoylethanolamide (OEA) and chylomicron synthesis, CD36 function in small intestine remains incompletely understood. Our in vivo data demonstrated that CD36 gene deletion in mice did not affect intestinal LCFA uptake. CD36 protein disappeared early from the luminal side of intestinal villi during the post-prandial period but only when the diet contained lipids. This drop was significant 1 h after a lipid supply and was associated with ubiquitination of CD36. Using CHO…

030309 nutrition & dieteticsCD36[SDV]Life Sciences [q-bio]030209 endocrinology & metabolism03 medical and health sciencesOleoylethanolamidechemistry.chemical_compound0302 clinical medicineIn vivoparasitic diseasesmedicineLipid sensorGeneral Psychology0303 health sciencesNutrition and DieteticsbiologyChinese hamster ovary cellSmall intestine3. Good healthCell biologymedicine.anatomical_structurechemistryProteasome inhibitorbiology.proteinlipids (amino acids peptides and proteins)CD36[SDV.AEN]Life Sciences [q-bio]/Food and NutritionEx vivomedicine.drugChylomicron
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Prevalence and clinical relevance of thyroid stimulating hormone receptor-blocking antibodies in autoimmune thyroid disease

2017

Summary The prevalence and clinical relevance of thyroid stimulating hormone (TSH) receptor (TSHR) blocking antibodies (TBAb) in patients with autoimmune thyroid disease (AITD) was investigated. Serum TBAb were measured with a reporter gene bioassay using Chinese hamster ovary cells. Blocking activity was defined as percentage inhibition of luciferase expression relative to induction with bovine TSH alone (cut-off 40% inhibition). All samples were measured for TSHR stimulatory antibody (TSAb) and TSHR binding inhibiting immunoglobulins (TBII). A total of 1079 unselected, consecutive patients with AITD and 302 healthy controls were included. All unselected controls were negative for TBAb and…

AdultMaleendocrine systemmedicine.medical_specialtyAdolescentendocrine system diseasesGraves' diseaseImmunologyThyroid Gland030209 endocrinology & metabolismCHO CellsHashimoto DiseaseThyroiditisYoung Adult03 medical and health sciencesCricetulus0302 clinical medicineCricetinaeInternal medicineBlocking antibodyPrevalencemedicineAnimalsHumansImmunology and AllergyEuthyroidClinical significanceAutoantibodiesbiologybusiness.industryChinese hamster ovary cellThyroidThyroiditis AutoimmuneReceptors ThyrotropinOriginal ArticlesMiddle Agedmedicine.diseaseeye diseasesGraves Diseasemedicine.anatomical_structureEndocrinology030220 oncology & carcinogenesisImmunologybiology.proteinBiological AssayFemaleAntibodybusinesshormones hormone substitutes and hormone antagonistsClinical and Experimental Immunology
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Cytogenetic effects of promutagens in genetically engineered V79 Chinese hamster cells expressing cytochromes P450.

1993

Abstract V79 Chinese hamster cell lines genetically engineered to express rat CYP2B1, CYP1A1, CYP1A2, and their parental cell lines V79-MZ, without acetyltransferase, and V79-NH, with acetyltransferase, were studied for chromosome aberrations and sister chromatid exchange induced by aflatoxin B 1 , cyclophosphamide, benzo[a]pyrene, 7,12-dimethylbenz[a]anthracene and dimethylnitrosamine. The parental V79 cell lines did not show clastogenic effects. Significant clastogenic effects were observed after an 18 h exposure to aflatoxin B 1 and cyclophosphamide in CYP2B1 expressing cells, to benzo[a]pyrene in CYP1A1 and CYP1A2 expressing cells, to 7,12-dimethylbenz[a]anthracene and dimethylnitrosami…

Aflatoxin B1910-Dimethyl-12-benzanthraceneHamsterSister chromatid exchangeMutagenToxicologymedicine.disease_causeChinese hamsterCell LineDimethylnitrosamineClastogenCricetulusCytochrome P-450 Enzyme SystemCricetinaepolycyclic compoundsmedicineBenzo(a)pyreneAnimalsCyclophosphamideBiotransformationPharmacologyChromosome Aberrationsbiologyrespiratory systembiology.organism_classificationPollutionMolecular biologyIn vitroRatsCell cultureAcetyltransferaseGenetic EngineeringSister Chromatid ExchangeMutagensEuropean journal of pharmacology
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Characterization of an epithelial, nearly diploid liver cell strain, from Chinese hamster, able to activate promutagens

1987

Epithelial liver cells of the Chinese hamster (CHEL cells) were propagated in culture for 35 passages. At favourable cell densities, the population doubling time in normal medium, was 20 h. L-Tyrosine amino transferase activity was retained at a measurable level, but its enhancement by dexamethasone was detected solely in cells of early passages. Pyruvate kinase was strongly activated by fructose-1,6-biphosphate at low substrate concentrations. These enzymatic properties suggest that the CHEL cells are derived from a sub-population of parenchymal hepatocytes or from cells closely related to parenchymal hepatocytes. With a lag period of a few hours, CHEL cultures metabolized benzo[a]pyrene. …

Aflatoxin B1910-Dimethyl-12-benzanthraceneHealth Toxicology and MutagenesisPyruvate KinaseCellToxicologyEpitheliumChinese hamsterCricetulusAflatoxinsCricetinaeBenzo(a)pyreneGeneticsmedicineAnimalsDoubling timeBiotransformationCells CulturedGenetics (clinical)Tyrosine TransaminaseGeneticsbiologyLiver cellEpithelial CellsMonooxygenasebiology.organism_classificationMolecular biologyClone CellsEpoxide hydrolase activitymedicine.anatomical_structureLiverKaryotypingPloidyCell DivisionPyruvate kinaseMutagensMutagenesis
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Stable expression of rat cytochrome P-450IIB1 cDNA in Chinese hamster cells (V79) and metabolic activation of aflatoxin B1.

1988

V79 Chinese hamster fibroblasts are widely used for mutagenicity testing but have the serious limitation that they do not express cytochromes P-450, which are needed for the activation of many promutagens to mutagenic metabolites. A full-length cDNA clone encoding the monooxygenase cytochrome P-450IIB1 under control of the simian virus 40 early promoter was constructed and cointroduced with the selection marker neomycin phosphotransferase (conferring resistance to G418) into V79 Chinese hamster cells. G418-resistant cells were selected, established as cell lines, and tested for cytochrome P-450IIB1 expression and enzymatic activity. Two cell lines (SD1 and SD3) were found that stably produc…

Aflatoxin B1CytochromeHamsterTransfectionChinese hamsterGene productAflatoxinsCytochrome P-450 Enzyme SystemComplementary DNACricetinaeAnimalsBiotransformationCells CulturedMultidisciplinarybiologyCytochrome P450TransfectionDNAMonooxygenasebiology.organism_classificationMolecular biologyRatsBiochemistrybiology.proteinMutagensPlasmidsResearch ArticleProceedings of the National Academy of Sciences of the United States of America
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Selection and characterization of a novel agonistic human recombinant anti-Trail-R2 minibody with anti-leukemic activity

2009

Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is a promising natural anticancer therapeutic agent because through its “death receptors”, TRAIL-R1 and TRAIL-R2, it induces apoptosis in many transformed tumor cells, but not in the majority of normal cells. Hence, agonistic compounds directed against TRAIL death receptors have the potential of being excellent cancer therapeutic agents, with minimal cytotoxicity in normal tissues. Here, we report the selection and characterization of a new single-chain fragment variable (scFv) to TRAIL-R2 receptor isolated from a human phage-display library, produced as minibody (MB), and characterized for the in vitro anti-leukemic tumoricid…

Agonistmedicine.drug_classTRAIL; TRAIL-R2; minibody; anticancer therapyImmunologylymphoma; therapy; recombinant antibodyTRAILApoptosislymphomaCHO CellsCricetulusPeptide LibraryTRAIL-R2CricetinaeImmunoglobulin FragmentmedicineAnimalsHumansImmunology and Allergyrecombinant antibodyanticancer therapyReceptorCytotoxicityImmunoglobulin FragmentsPharmacologytherapyLeukemiaChemistryAnimalChinese hamster ovary cellAntibody-Dependent Cell CytotoxicityminibodyApoptosiIn vitroRecombinant ProteinsReceptors TNF-Related Apoptosis-Inducing LigandCHO CellCell cultureApoptosisImmunologyCancer researchTumor necrosis factor alphaCricetuluHuman
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